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PenDimcns onanalysis simciy (Ccn Inclusion Qjualion Allor Fohina Lio -ulion Inin DT ansiomlaniys Cn Usuxt I0 sot up %ho convorsion lactcrs Iht lead t0 desred valuas within Itas Map Tne correci serp 0l 47 equntka Can vorilied LY chocking d tne resull Kill only have Ino desirod Urnts arot unit careulallon3NAClaq ) AI(NO-h(aql- AICL () 3NANO-(#q) For Ihi doechIan [CACmn voihava Ae0 L soliaan D#do NaCllaq) Uunatna tulio map Irom Pad ramnleto Iho Iollnnn dunwinnl ana '5/5 Inal aldaSYjuto C Qulal

Pen Dimcns onanalysis simciy (Ccn Inclusion Qjualion Allor Fohina Lio -ulion Inin DT ansiomlaniys Cn Usuxt I0 sot up %ho convorsion lactcrs Iht lead t0 desred valuas within Itas Map Tne correci serp 0l 47 equntka Can vorilied LY chocking d tne resull Kill only have Ino desirod Urnts arot unit careulallon 3NAClaq ) AI(NO-h(aql- AICL () 3NANO-(#q) For Ihi doechIan [CACmn voihava Ae0 L soliaan D#do NaCllaq) Uunatna tulio map Irom Pad ramnleto Iho Iollnnn dunwinnl ana '5/5 Inal aldaSYjuto C Qulalo maa; ol AICL(s Ihai can lormoo Dacino Ino vallJS pch cony05on (jc % accEdina wno h 0rthoy shalld acocdan Iro Nimamic dnnonmon Drag tne npproprlato valuos Inalt rogpo cilyo Inrdota VlnvAuallhlc Hicua! 0,058 Itlal Aich( ) J uuol NaCier ) Etaul AICL (s) 0.525 Iuol AICh() 0.037 ItalAICL(0) 0 13n mol ACH(s) 0 T40 L Bulian 0,Bc0 mol NaCUnq) Gup Croud 2 Omdb Qmut Doup Gmud ? scil Aohaln



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Flight formula for Indian spotted owlets The following table shows the average body mass $m(t)$ (in $g$ ) and average wing chord length $w(t)(\text {in } m m),$ along with the derivatives $m^{\prime}(t)$ and $w^{\prime}(t),$ of $t-$week-old Indian spotted owlets. The flight formula function $f(t)=w(t) / m(t)$ which is the ratio of wing chord length to mass, is used to predict when these fledglings are old enough to fly. The values of $f$ are less than $1,$ but approach 1 as t increases. When $f$ is close to $1,$ the fledglings are capable of flying, which is important for determining when rescued fledglings can be released back into the wild. (Source: ZooKeys, 132,2011 ) $$\begin{array}{lrrrr} t & m(t) & m^{\prime}(t) & w(t) & w^{\prime}(t) \\ \hline 1 & 23.32 & 41.45 & 10.14 & 14.5 \\ 1.5 & 50.59 & 64.94 & 20.13 & 26.17 \\ 2 & 82.83 & 57.95 & 36.7 & 39.86 \\ 2.5 & 105.13 & 31.08 & 58.92 & 47.11 \\ 3 & 115.48 & 12.48 & 81.55 & 41.38 \\ 3.5 & 119.4 & 4.44 & 98.99 & 27.94 \\ 4 & 120.76 & 1.51 & 109.75 & 15.74 \\ 4.5 & 121.22 & 0.51 & 115.5 & 7.99 \\ 5 & 121.37 & 0.17 & 118.34 & 3.85 \\ 5.5 & 121.42 & 0.06 & 119.69 & 1.8 \\ 6 & 121.44 & 0.02 & 120.32 & 0.84 \\ 6.5 & 121.45 & 0.01 & 120.61 & 0.38 \end{array}$$ State the units associated with the following derivatives and state the physical meaning of each derivative. a. $m^{\prime}(t)$ b. $w^{\prime}(t)$
c. $f^{\prime}(t)$

There is a kind of bacteria called yersinia pestis, and this actually is a bacteria that lead to the black plague or just the plague. And it's an extremely violent kind of bacteria. And so once a person has been exposed to this bacteria, the microorganism injects a set of effective proteins into part of the immune system. The macrophages, the big cells that eat things that are not self. But they inject these effective proteins, and it ends up causing a problem that they can no longer go around and try to eat the invading germ cells. And so it interfered with our ability to have been in eight and in response. And so what happens is that these effective proteins and there's one specifically called Y O. P J or Bob Jay. What it does is that the BOJ blots, the Ta K one signaling pathway and what the TA came on. Signaling pathway mhm is that it's a key civilian step in the innate immune response so that the microphone versions are able to chop down and break apart the bacteria. And so what the what Jay is doing is it will assimilate false related Syrians and three innings serene and three inning are both immuno acids, and since it's assimilating these things, it's causing a slight change in their structure. And so it causes a structural change. And, uh, yeah, sort of protein, that is. Oh, one of the map kinase is, uh and so and what it is, It's the specific, um mhm. So it ends up being a problem in terms of the kind of protein for the PH. A woman and the teen AK one. Since it's not able to be functional because its structure is different, since the Syrians and the trainings have been assimilated uh, the action or the activity in terms of how is it interfering with Ta K one is it's causing the deactivation of P a k one Mhm, Yeah! Mhm, Yeah, yeah, Mm hmm.

He's a kind of bacteria called yersinia pestis, and this actually is a bacteria that lead to the black plague. Just the plague. And it's an extremely violent kind of bacteria. And so once a person has been exposed to this bacteria, a microorganism injects a set of effective proteins into and part of the immune system the macrophages, the big cells that eat things that are not self. They inject these effective proteins and it into causing a problem that they can no longer go around and try to eat the invading, uh, germ cells. And so, uh, it interferes with our ability to have an innate in the English response. And so what happens is that these infected proteins and then one specifically called Y o P J object. But what it does is that the watch Jay bless the Ta K one civilian pathway and what the TA came in Signal pathway mhm is that it's a key civilian step in the in the immune response so that the microphone versions are able to chop down and break apart the bacteria. And so what the what Jerry is doing is it will assimilate fast forwarded, see rings and three news. Serene and three need are both of, you know also. So if you look and see below, these are phosphor related, um, serene and three innings. And what the op J does. Is it a silly dilates? And I'm just doing a representation here that it changes the shape of the protein by adding and, uh, doing this assimilation. And by doing that, it makes the protein makes the amino acid the singular amino acid of the three in in the singular amino acid of the Syrian change in the shape of it, and make him so that this whole entire protein is non functional. Yeah, for instance, it's assimilating these things. It's causing a slight change in their structure. And so it causes a structural change. And, uh, sort of protein that is Mm uh, one of the map kinase is of, and so and what it is is it's the specific Oh, and so it ends up being a problem in terms of the kind of 13. But the Ta K one and the TA came one, since it's not able to see functional because its structure is different since since the Syrians and the three needs have been assimilated. Uh, the action or the activity, Uh, in terms of how is it interfering with Ta K? One is It's causing the deactivation of ta k one. Yeah, mhm. So it's no longer active. It's become deactivated. Mm. And this is stemming from the bacteria y pestis injecting its effective proteins into the bacteria, I mean, into the macrophage into the human immune cell. And so we no longer have the ability to have the developed in eight. This is in terms of the human cell that we no longer have the innate immune system. Mhm. Yeah. Yeah, because it changes. It brings in. We no longer have that because it brings in this assimilation. And it attaches to those foster related Syrians and theory. Three innings of the amino acid sequence

Mhm. There's a kind of bacteria known as you're saying pestis, and it is actually the cause, the black plague or just the plain. And it's extremely, very little kind of bacteria. Yeah, mhm. Once a person has been exposed to this kind of bacteria, the microorganism actually injects a set of effective protein into the macro for just the big ears. This is part of the immune system, um, what they call the innate immune system. And it's part of the immune system that recognizes itself. Nonsense, and these particular effect of proteins are injected into the big yourselves. It ends up causing a problem and then the macro fishes and no longer try to eat up the germ cells. And so it interferes with a signaling pathway of 18 AK one. Ta K one is actually a major key signaling step in the innate immune response pathway. And so if these effective proteins that are coming from the y pestis thing that causes the plague, essentially, it's going to interfere with the th one and therefore interfere with the innate immune response passport. So what happens is that these defective proteins and one specifically is called the O J on y O. P and then a capital J. And what happens is the yacht Jayne blocks the ta K one signaling pathway. Yeah, What happens with this signaling pathway is there is a key signaling step so the macro fires can identify and make sure that they're in and the things through the not self, as opposed to the things that herself. And so what they captured does is it actually assimilates syringe and grenades. Which arm, you know, assets found on the ta K one see rings and green beans are two different kinds of amino acids, and they assimilate these and they cause a change in the structure, which a change of the immunized structure, or the way that it's shape is, is going to change the shoot, the structure of the protein overall, making this Matt Cain's a T a K one also have a change in the structure and no longer do a specific job. And it continues to be a problem for that particular kind of protein that he came one. Since it's not able to be functional because the structure is different. Since the Syrians and three innings have been assimilated, the activity of in terms of how does it the activating blocking the TA K one is by changing the structure today. Trade One. Okay, the TA K one of the macrophages, A map kinase murkiness says, Mm hmm. Um, that's a particular style or particular kind. And this particular protein is the one that will go out and allow for the macrophage to eat up again because it's macro big, and then the polish eat so it eats the big things, or it's a big sell that eats things. And because it is, um, got a different structure because of the symbolism that I put on my list here. The serene, um, trip to fan losing three. And I mean it is no longer functional. The map can ease the protein of T T. A. K one is no longer active. It has become deactivated. And since it's deactivated, we will no longer have the ability and the human cell to be able to have the innate immune response. It won't happen. Mhm. Okay,

Yeah, your city a pesticide is a bacteria, and I have on our screen a little cartoon representing a kind of a bacteria. And anyway, this is the bacteria, though that leads to the black plague, which, as you remember from your history, caused the little dots the post, um, places and was spread by rats in Europe. And we've lost, um, so many people during that time. It's also called Just Played for Short. It's an extremely virulent kind of bacteria, and in fact, they had what they called plague doctors. If you recall again from your history, Um, once a person has been exposed to this kind of bacteria, the microorganism, which is in this image here there's two of the images. They're both somewhat drawn, and this looks like it's going through binary fission. But once a person has been exposed to or is, um, having their cells, which this is a little cops. L like a white blood cell. What they talk about it being a macro Farge. And it's the same as this green colored in the image below. Once a person has been exposed, the microorganism objects a set of effective proteins into the macro fagioli macrophages known as the big eater of the So, and it's part of the immune system, and it's called the innate immune system. It's a part of the immune system that recognizes self from non self. These particular effective proteins are injected into the big eater cells, and it ends up causing a problem and that the macrophages can no longer actually try to eat up those germ cells, as in the image here, to the right, there's a bacterial cell that's connected with that macrophage, and part of the job of the macrophage is to eat as a big sell eater or a big eater right in the text next to the right of the picture. Um, is that the effect of proteins once again inject into the macro? The macro is big, and footage is eater. Yeah, and so, um, ego psychosis is when the cell is eating. Um, and so anyway, that's where we come up with the word macrophage. Macro big. It's bigger than any of the other cells here, and it's eating that germ cell. And here is a picture, then showing the process of a big eater sell. Here's its nucleus and there's a germ cell or some kind of bacteria, and it's eating it and breaking it apart. And so that's its job. And what happens when the why prostatitis, um, injects its effective proteins? It disrupts that process. There's a signaling pathway called ta K one. Ta K one is a major key signaling step in the innate immune response pathway. And if these affect our proteins are in fact, um participating and, um, disrupting, which here is an image of the T A k singling one pathway being disrupted by the injected proteins on the part of the Y pestis. The Yap J is in fact, the protein that is the one sent by the bacteria. It's the one that the effect of protein that I was talking about and it assimilates any of the Syrians or three innings of the different kinds of amino acids in this ta k one. And so what happens is, um, this yeah, J, which is a capital y and then, uh, lower case O. P and then a Capital J is that the Jac J actually blocks the D a k one signaling pathway. Yeah, and, um, again that it assimilates it attaches to, and it changes the shape of any of the sufferings in any of the three innings that are there on the T a k one map kinase. So one of the things that I wanted to point out is, what are amino acids? Specifically, what is the Syrian and the three inning? So amino acids are the different kinds of macro molecules that are in any kind of living thing, and that they joined together to make the polyp uptight or make the protein. And in this case, it's the Paula peptide, or protein that's at the ta Ke, signaling one pathway. And this is an example. This is an actual three D structural molecule, structural shape modeling for the searing, which is one of the immuno acids, and the red over here is a carb oxalic acid. And then over here is a three inning, and when the yup J from the why past itis is in fact injected into the macrophage along the signaling pathway, these amino acids, these two specific amino acids, are assimilated. Here is a saddle, So instead of it being carb oxalic acid over here on this other with the training camp oxalic acid. And instead we have an acetyl group where the structure of the amino acid is being structurally changed, its being altered. So on the Syrian, we're getting this additional, um, shape. I'm just going to add this little icon here and on the three inning, we're getting this additional little shape. And instead of having the carb, oxalic acid and having it being foster related, it's now got this. A Seattle? Yeah, a C E t. Why l And since it's now got this a Seattle, um addition and change instead of it being it's normal amino acid structure that it's supposed to be. It is now no longer functional, and you know, so if you were to continue on in the amino acid string, any of the other kinds of Syrians and three innings are also equally affected. And so it causes this problem, and it makes it to where that since their structure, their shape is then going to then change the structure of the protein overall. So it makes this map kinase. This kind of protein that's in this Ta K one signaling pathway also have a change in its structure, so it's no longer able to do a specific job, and it continues to be a problem. So then, since it's not able to be functional, which is to answer the question more directly, since its structure is different from what it was supposed to be, literally in the three dimensional space, the Syrian and the training are differently shaped as a result of the assimilation that has happened. Then we have it being deactivated. The activity of this particular protein is deactivated or is being blocked because of the change of the structure of the TA K one. And so, since the change of the structure happens, there's no way that the signaling pathway can take place and the macrophages no longer able to be that big eater, is no longer able to eat the virulent bacteria from the, which is called the Y uh, tests, which is that one that's led by the causes black


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