In order to answer this question, let's write down some facts. We know from careful reading of the question that the rabbits that were normal were true breeding. So that means that they have a homeless IBIs state. And so that's a key piece of information for us to solve this problem. And so let's work on a part. A part told us that of the offspring, there were 217 helder anomaly rabbits, and there were 237 normal rabbits, and this is a 1 to 1 ratio. And so, if we think about okay, what were the parents of this off spur of thes offspring in the F one? One of those parents have to be home is itis, so they have to be. Either parents were either big a big A or little, a little A because that's you. That's the definition. True breeding is that normal produces always normal offspring. That tells us that Helder must be headers, I guess because they're different than the normal. But the normal rabbits can only donate one normal alil, and so we know that normal is true breeding. So either big a big a or a little, a little a must be normal. And the headers I go Must be pulgar. Okay, so then let's work through Be part deep Heart says what happens basically if you breathe a poker rabbit with another Kulbir rabbit Well, and so we know that they're headers, August So it's essentially a a situation like this, And here we can do a simple mono hybrid cross and we can put our Leal's from the appropriate windows, and then we can bring them together to represent the offspring. And so when we do that, we know that Helder is going to be the headers idea. So here is the helper, and then one of these is normal, and I'm not sure right now we have enough information to tell, So you're just gonna have to choose one. Um And so I know that helder a wheels are recessive So, um, we know, at least in humans. So here we get 223 normal offspring and this is in the problem. 439 helder offspring and 39 very abnormal. Probably won't make it toe birth if they were born. They die shortly after because they have extensive deformities. Um, and so here, if we look, this is a 1 to 2 to one ratio, and that's what we expect from a breeding between two headers. I get individuals. And so, um, we know that this abnormal one here is lethal. And in humans, those things don't make it to term usually. And so here we have the predicted ratio, where big a big A will say is normal and a little, a little a is lethal and that you expect 2/3 of your surviving offspring to exhibit the abnormal white blood cells, which is characteristic of the pulgar alil. But it's not lethal. Um, because it's not in the home was like estate, so 1 to 2 to rate one ratio and then to answer C part. Since we know a little bit more, they saw the ratios from the problem. In our information, we have that the Pilger polio is a true pick in that In the hitters, I get condition you have a dominant alil that's expressed that produces segmentation of the nucleus and white blood cells. And remember, white blood cells are part of the immune system, and they're responsible for defence against pathogens and foreign particles and in the home is like a state. So little, a little A. It is a lethal condition that organism doesn't or that individual doesn't make it for very long. In life, it's it's born at all. And so, um, if you wanted to investigate Maura about the inheritance pattern to confirm or refuse our hypothesis that when homes I guess recessive conditions are present, its lethal you could do like a statistical analysis to compare litter size with normal versus the elder animals and things like that, um, so that you can determine, um, whether our hypothesis of legality is supported or refuted. And so if we take that information and apply it to the human population, we don't see Pell Grant abnormality very much. If it all, it's extremely rare, and that's because it's a recessive lethal. And so what happens as a recessive lethal in the human population? That means that one in 1000 people have a little A and so if you take too, you know, parents who have big a little a big a little a, and they have a baby that has the lethal condition that's a one in a 1,000,000 chance. And then if you have one out of 1000 random matings that this is gonna happen, then that sort of is to still down toe one case in four million people. And so the baby who has this condition probably doesn't even make it to term. So it's unlikely that the baby even is form. It's probably going to lead to a miscarriage. And so the conditions extremely rare. It probably goes unrecognized in, um, the population. And that's why there's this absence of the public anomaly in humans because it's a recessive, lethal polio, and it's extraordinarily rare. So if we do the cross to support this, what we have is going to a simple pennant square, and we can put our wheels on the appropriate windows, and we can use the information that we've talked about to answer the question and some just bringing these together. And so here we have one. And it's tricky because you don't count the recess of the process of lethal Leo. So it's 1/3 big, a big a normal, and 2/3 will have the Pilger an anomaly. And then this little a little way You don't count because it's lethal. And so, if you look at, um, the anomaly 2/3 chance that they'll have the anomaly. Um, but some of them die, and so they don't survive until birth.