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Use the_ following information to answer the next question Role of Calcium Ions in the Contraction of # Myofibril Calcium ions bond to protein molecule called tropo...

Question

Use the_ following information to answer the next question Role of Calcium Ions in the Contraction of # Myofibril Calcium ions bond to protein molecule called troponin that is part of the actin myofilament The attachment sites for the myosin heads are physically blocked by protein called tropomyosin The myosin heads can bind t0 the actin, and contraction occurs The : calcium ion/troponin complex exposcs the myosin binding sites of actin80 When these steps are placed in the correct sequence of ev

Use the_ following information to answer the next question Role of Calcium Ions in the Contraction of # Myofibril Calcium ions bond to protein molecule called troponin that is part of the actin myofilament The attachment sites for the myosin heads are physically blocked by protein called tropomyosin The myosin heads can bind t0 the actin, and contraction occurs The : calcium ion/troponin complex exposcs the myosin binding sites of actin 80 When these steps are placed in the correct sequence of events the order of this sequence would be: 2413 1243 1234 2143



Answers

3. The release of calcium ions initiates muscle contractions. Watch this video (http://openstaxcollege.ore) calciumrole) to learn more about the role of calcium. (a) What are "T-tubules" and what is their role? (b) Please also describe how actin-binding sites are made available for cross-bridging with myosin heads during contraction.

This question. We've been given a video of calcium islands in muscle contractions were getting some questions about it. So a what are T tubules on? What is their role? So TT rooms are extensions of the soccer member, so extension and their role is to forgive a release of B County. Miles from the socket socket class in particular are so it when you need it, actually essential when it reaches in your muscular junction of the teacher abuse was the release of the county Morals way have be on the B were describing how acting bonds insights are available, prospecting with less intense. So this is the role of council miles. Be calcium ions C 80 plus binds to fruit Morrison and these ferpa Marcin proteins are phones attached to the binding sites on the accent. But when counselling bonds to them, beetroot Marcin moves and because it's moving away from binding site, the binding site is now physically available to cross bridge with the Myerson

Already. So this question is talking about the Sarko plasmid particular and how uh, signaling takes place via calcium. Um, changes in calcium concentration within our muscle cells in a muscle contraction on. So we'll start out by, um, diagramming out our Sarko plasmid particular here. So imagine this is our Sarko plasmid particular membrane. So this is going to be the inside of the cycle plasma particular, um, on this side. So our Sarko plastic particularly there's a whole bunch of calcium stored up in this Sarko plastic particular here. So the calcium is this blue dot all these blue dots here, the question is asking us why is it important that the channels that release the calcium into the site the site is all the self? So the site is always gonna be on this side. Why are these channels that release the calcium into the site is all the cell? Why is it important that they are, um, that they receptors for opening Art is calcium itself. Right? So I'm gonna have these channels here in the cell. And when a deep polarization event occurs, um, what's gonna happen is some of this calcium is going to rush out, um, into the site is all that's gonna allow our muscle to contract. And however, some of this calcium is going to buy into this channel here, and that's actually going to, um, open mawr channels along the membrane here. So all these channels in the membrane have the capability of binding to calcium. And so the more calcium out here in the sight is all the more channels they're gonna open right, because this calcium can now bind to these channels. And so the mawr influx of calcium we have into the, um, side is all and so this is a positive feedback loop, and this is going to ensure that we can have a strong and unified muscle contraction because all this calcium is gonna rush out all at once because it's a positive feedback loop. Is opening Mawr channels as it comes out on DSO. That's really important because we want to contract our muscle fiber all in one motion. We don't want to be a slow process. We want to be a extremely quick

Okay, So for this particular problem, we are looking at a process. And we're trying to order the events that take place for this process to be completed. So this is a brief overview of ah process known as muscular contraction also can be referred to his sliding filaments theory for the sort of end portion off this process so abbreviating at the top here with EMC for muscular contraction. So this is a voluntary process, meaning we needed a nervous impulse. So signal from the brain or spinal cord for this process to begin and its preliminary stages, which we see here This is a rough drawing of an Exxon. An action potential is going through this Exxon, which is a portion of a neuron when we have this specified with the and in parentheses for neuron. Ah, this will reach the end of the Exxon terminal releasing a subtle Colleen into the synaptic cleft. So were the most were the neuron is snap sing with the actual muscle. So Aziz so we need toe first understand that this will be taking place first. Ah, it's ah necessity for the action potential to travel throughout the neuron giving an impulse for muscle contraction to begin in the first place. So that is first. So after this we have this year, Colin, which will bind this, will constantly cause an action potential in the muscle. So we see this outlined here with a P for action potential muscle in parentheses, and this will eventually lead to some, uh, some action occurring in the circle. Plasvic Ridiculous as our revie ated here. And this will release calcium from the circle plastic particular and calcium has an important function here, so we'll go over that in a second. But first we need to understand that it's actually potential travel to cause this Cirque applies in particular to release calcium. So in this calcium, it goes somewhere and it will end up binding to trope in in abbreviated here. And when it binds, it causes movements. Thio expose ah portion off the acting filament. So acting is exposed so that my chosen combined to acting because acting amazing, they bind together. Specifically, mirelson will bind to the accident, and this is causing the sliding filament theory, which is ultimately what causes muscle contraction at the end of the day. So we recognize the SAARC applies in particular releases calcium. Then the calcium binds atropine in. And then my accident acting can perform the sighting filament. Ah, portion off muscle contraction. So the way it's laid out in the question action potential is what's it is number three, So that will be first. After that, we have start cold plasma particular releases. Calcium that's listed is one. So we're three 31 is based on the reading of the question, not to the specific order, but three is referring to what happens first and so on and calcium binding. A troponin is referred to as the fourth, and then my son binds to act in as referred to as the second. So this would be the particular order as the question, um, specifies. But we don't need that. That is the actual sequential order of things.

You're in contraction. What causes be cross bridges between the mice and Jacksonville mints To detach a myson heads binds to ADP be myson has monster a teepee See calcium ones tripping in Was he o si bonds too active? So the answer here is be when Merson has once to 80 p and be recently this is detachments on the movements of the fibers alongside each other Be filaments so excited requires energy and this energy is provided by 80 p and this finding to my city.


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