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Is p89 tumor suppressor Or proto-oncogene? Explain your answer thoroughly...

Question

Is p89 tumor suppressor Or proto-oncogene? Explain your answer thoroughly

Is p89 tumor suppressor Or proto-oncogene? Explain your answer thoroughly



Answers

What is a tumor suppressor?

Here first of all, asked what a tumor suppressors. So a tumor suppressor here is a molecule that restricts the ability of a Celtic grown divide, and oncogene on the opposite hand is going to have the opposite effect where it stimulates the Celtic grow. Divide about the tumor suppressor here is going to restrict.

Here once it's described the difference between a tumor suppressor and on co gene with relation to the actual cause of cancer. So a tumor suppressing here is a protein that helps control salt growth. So it control cell growth and division, so acts like the brakes on a car. On the other hand, here, the oncogene is a particular opposite effect of a tumor suppressor, so an oncogene stimulates cell growth and division and access the gas pedal to a car.

There are a number of tumor suppressor genes we want to know which is not one of them. Mhm R E T is not a tumor suppressor gene. The art gene provides instructions for producing a protein that is involved in signaling within cells.

This question is asking us to compare proto uncle jeans two tumor suppressor genes An important component to understand the difference between these two is to remember that humans have 23 pairs of chromosomes. So on each chromosome we have two copies of similar genes that will be found on each pair. So proto uncle genes We can break this apart pro meaning before and oncogene is going to be a gene promoting cancer. So these are jeans that are going to basically caused the cancerous formation inside a cell, so these genes are going to typically transcribe proteins necessary for cell division. This can happen on just a single mutation of one of our chrome attitudes. So say we have two copies of a certain gene of chromosome wine, and this will occur on only a single promoted. This cremated will be able to produce that protein that will cause that cancer. So there are many different examples of this. You have what's called a B, C R A B l. And this is a fusion of two different chromosomes, and this is going to allow for the uncontrolled growth of the cell because it's allowing a growth factor signal to constantly be transmitted to the cell. This is opposed to a tumor suppressor for this mutation typically has to occur in both copies of the chromosome. So in this case, you're going to have your chromosome. This will be one, and this is typically a knockout, so it's eliminating a regulator. So you're comin. Regulators. You have the retinoblastoma gene, and you also have p 53 are very both very important regulators. So when you have two copies of the same cremated, we're going to say this is our retinoblastoma protein gene. However, this copy is going to appear on both of those chrome attitudes. So if there's a mutation in one of the crow motives, you're still going to be able to make that retinoblastoma protein from that healthy chroma tube. However, a tumor will occur when you knock out both copies. So here, in this case, we're saying you have to mutations so you won't be able to make the retinoblastoma from this promoted, and you won't be able to make the retinoblastoma from that chromatic. So in this case, you would not be making your retinoblastoma. So this these are regulators of the cell growth and cell division. I think, and as long as you have one healthy promoted able to produce one of these proteins, it should allow the cell to function normally. However, these will require that double knockout in order to cause that cancer. So the differences between these two are proto uncle jeans. Typically are mutations that cause and make proteins that allow the cell to grow, divide and had those abnormalities, whereas your tumor suppressors typically eliminate the production of proteins. I must occur in both copies of the chromosomal gene, and these will typically be in regulators such as retinoblastoma, or P 53.


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