Yeah, mhm. And what's happening with the salmonella is at first stimulates the membrane in order to promote invasion and then suppresses the membrane once the invasion is complete. So there is another set of proteins. There's actually two protein that will help or mediate this process by having their proteins being injected. S o k e. And that's the one that promotes that helps the membrane invasion to happen. And then SPT capital P SPT Capital Pain, which then blocks the effects of s okay, and so that would then sort of be the end part of it where it suppresses the membrane roughly is what they call it. R u F f l i N g Once the invasion is complete. So the question is, First of all, how does the S o p e n s p t p effect? The Mon America G T P A s, which is called capital, are lowercase a C capital R A. C in its active form is what is going to be promoting for the membrane. Roughly. How does this affect the R A C activity? Um, okay. And another question it says in here is How do you suppose the effects of the S, O P E and the S P T P R staggered in time. How does that take place? That they are staggered, that there is one that's happening at one point and then later on in time there's another thing that's happening. But then the answer to the first question that s O P e is, uh what's gonna activate and get the RS seem to get started? And so what we're going to have to have happened here is at because GDP disassociate it's really pretty slowly we're gonna have fall. Hey, some help. I'm doing that GDP to be disassociated from GPAs and then instead of the GDP being there, we'll have GTP, and, uh, volume of the G t. P in place of the GDP is what's the activation? Um, it stabilizes the GTP interactions and then allows for basically the guarani in exchange if you will, and it activates the g t. P. A s in the order for the r i C to do the membrane ruffling Now the S P t. P is called a gap G a p, which stands for octopus Activating. Hello, chief. And what it does is it cleaves off GTP away from the GTs and renders the RSC inactive. And so since the RSC is inactive, then the ruffling of the membrane is inhibited. It stopped. And that's once the invasion has been completed. Honest, that is there. So there needs to be a little bit better in terms of how the S O, p. E and S p p affects the R I C activity and, um affecting this modern American beauty P a s. And I'll explain that in just a minute able to stagger this stuff, how the proteins are able to be centered, it was because of having different rates of breaking down the S okay, a degrades way with much sooner. And then the S P T. P actually sticks around and is broad persisted for a longer period of time after being injected. So the SNP was the one that initiated the promoting of the membrane raffling invasion, and so integrates first, it just simply breaks down faster. Whereas the S P T. P stays around is more persistent for a longer period of time. And it's the one that is, um, blocking the effect of the s O p. A. As well suppress the membrane roughly once the invasion is complete. So then back to the first question, and that first question was Answer it already. So hopefully we have a good idea about how this overall whole entire answer needs to be done.