Okay, So from 20 is all about the, uh, my Trojan activated protein kinase cascade. Right? And so these are triggered by r r t k receptors. So those those with an intra and like, extra sailor component, Okay. And it's what's gonna happen. Is the the M a p K pathway. Basically, that sort of has this r e S r a f This is a whole cascade, right? Going threes. Proteins here, uh, the any K protein on dhe, the e r k protein. And this this cascade this phosphor relation cascade. Uh, this, You know, this is also sort of called this, uh, this It is a parent to see this map. Okay, pathway. This is what will trigger cell division. Right and subdivision equals equals growth. So this is gonna because growth or division however you want to. I think about it, okay? And so we have a list of over what would cause uncontrolled growth. Right? And so we want to see what sort of mutations would cause more of this to happen. That's just looking through, uh, all these options. I think sometimes it's easier to pick out the things that are in incorrect right. So, for example, one of the items listed under a is a loss of function in the, uh, map ke pathway. Right? So if we have a loss of function of our map ke Okay, well, so that would be this not functioning. Okay, but that doesn't function that causes growth. So there's no way that that mutation would lead to uncontrolled growth rate s. So then we know that a a can't be one of our answers, right? And so if we look at, then if you look it see, be here one of the ones listed and b is to have the eye cap of the Greek happe here, uh, be permanently bound to the and F Kappa. Be no. Remember the eye Kappa B is an inhibitor of the nf Capa be. And this this guy will, uh, lied to sort of starts. You're transcription of your DNA, right? So this this is gonna transcribe. And so if this is currently inhibited, it could never enter the nucleus and start that process. You certainly need more deanna to duplicate, right? So this, uh, the eye Kappa be permanently bound would would would certainly not allow for for, uh, this additional and extra growth. Okay. And then if we want to take a look at our choices in D Okay, so this one has a loss of function. Mutation is well in both the r s and R f. Right, But we need those. We need those to get to this growth and division. That's part of our our map. Cape halfway. Okay, so this this r E s slash r a f protein lost would not lead to more growth. Okay, so it sort of just leaves us with, see, right. But if our RS protein was unable to head drawl size, then it's not gonna sort of do it needs in the cascade. And if we have loss of function for the loss of function of of the inhibitor, right, then our NF capa be will always be able to enter, uh, the nucleus. So we would we would sort of get that function back. So that's that checks out. That's good. Uh, if we gain a mutation in the genes for the pathway so there's gain of function somewhere in here, that means it's more functional and creating more growth division that checks out that would certainly help. And then, lastly, an unregulated phosphor relation. Cascade writes that this cascade is sort of happening in here. And if that's unregulated, that means that it has the potential to sort of grew out of control and not check itself. Um, so So all the all the options here and see do seem to check out, and that is our answer is letter C.